Intestinal Tumorigenicity of Mice Carrying Hemizygous Pax6, Pax6Sey-4H

Author:

NITTA Yumiko1,YOSHIDA Kazuko2,NAKAGATA Naomi3,NITTA Kohsaku4,HARADA Toshihide5,ISHIZAKI Fumiko6

Affiliation:

1. International Radiation Information Center, Research Institute for Radiation Biology and Medicine

2. Radiation Hazards Research Group, National Institute for Radiological Science

3. Center for Animal Resources and Development, Kumamoto University

4. Senogawa Hospital

5. Department of Health Services Management, Hiroshima International University

6. Department of Health Science, Hiroshima Prefectural University

Publisher

Japanese Association for Laboratory Animal Science

Subject

General Veterinary,General Biochemistry, Genetics and Molecular Biology,Animal Science and Zoology,General Medicine

Reference12 articles.

1. 1. Alexander, B.J., Rasko, J.E.J., Morahan, G., and Cook, W.D. 1995. Gene deletion explains both in vivo and in vitro generated chromosome 2 aberrations associated with murine myeloid leukemia. Leukemia 9: 2009-2015.

2. 2. Hayata, I., Ishihara, T., Hirashima, K., Sado, T., and Yamagiwa, J. 1979. Partial deletion of chromosome 2 in myeloid leukemias of irradiated C3H/He and RFM mice. J. Natl. Cancer Inst. 63: 843-848.

3. 4. Lyon, F.M., Bogani, D., Boyd, Y., Guillot, P., and Favor, J. 2000. Further genetic analysis of two autosomal dominant mouse eye defects, Ccw and Pax6coop. Mol. Vision 6: 199-203.

4. Spontaneous and Radiation-induced Leukemogenesis of the Mouse Small Eye Mutant, Pax6Sey3H

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