Mito-mice∆ and mitochondrial DNA mutator mice as models of human osteoporosis caused not by aging but by hyperparathyroidism

Author:

Mito Takayuki12,Tani Haruna3,Suzuki Michiko3,Ishikawa Kaori13,Nakada Kazuto13,Hayashi Jun-Ichi4

Affiliation:

1. Faculty of Life and Environmental Sciences, University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8572, Japan

2. Japan Society for the Promotion of Science (JSPS), 8 Ichiban-cho, Chiyoda-ku, Tokyo 102-8472, Japan

3. Graduate School of Life and Environmental Sciences, University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8572, Japan

4. University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8572, Japan

Publisher

Japanese Association for Laboratory Animal Science

Subject

General Veterinary,General Biochemistry, Genetics and Molecular Biology,Animal Science and Zoology,General Medicine

Reference25 articles.

Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Mitochondrial Genetics and Function as Determinants of Bone Phenotype and Aging;Current Osteoporosis Reports;2023-08-05

2. Research development and the prospect of animal models of mitochondrial DNA-related mitochondrial diseases;Analytical Biochemistry;2023-05

3. Mitochondrial DNA Mutations;Encyclopedia of Gerontology and Population Aging;2021

4. Metabolic reprogramming and the role of mitochondria in polycystic kidney disease;Cellular Signalling;2020-03

5. Mitochondrial DNA Mutations;Encyclopedia of Gerontology and Population Aging;2019

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