WBN/Kob-Ht Rats Spontaneously Develop Dermatitis under Conventional Conditions: Another Possible Model for Atopic Dermatitis-Reference-Cited by-同舟云学术

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WBN/Kob-Ht Rats Spontaneously Develop Dermatitis under Conventional Conditions: Another Possible Model for Atopic Dermatitis

Published:2005 Issue:5 Volume:54 Page:461-465
ISSN:1341-1357
Container-title:Experimental Animals
language:en
Short-container-title:Exp. Anim.

Author:

ASAKAWA Makoto¹,YOSHIOKA Takeshi¹,HIKITA Ichiro¹,MATSUTANI Takaji²,HIRASAWA Tsutomu¹,ARIMURA Akinori¹,SAKATA Tsuneaki¹,HORIKAWA Tatsuya³

Affiliation:

1. Shionogi Discovery Research Laboratories, Shionogi Co., Ltd.

2. Division of Immunology and Embryology, Department of Cell Biology, Tohoku University School of Medicine

3. Division of Dermatology, Department of Clinical Molecular Medicine, Kobe University Graduate School of Medicine

Publisher

Japanese Association for Laboratory Animal Science

Subject

General Veterinary,General Biochemistry, Genetics and Molecular Biology,Animal Science and Zoology,General Medicine

Link

http://www.jstage.jst.go.jp/article/expanim/54/5/54_5_461/_pdf

Reference19 articles.

1. Locus of Dominant Hairless Gene(Ht) Causing Abnormal Hair and Keratinization Maps to Rat Chromosome 10.

2. 2. Bunikowski, R., Mielke, M., Skarabis, H., Herz, U., Bergmann, R.L., Wahn, U., and Renz, H. 1999. J. Allergy. Clin. Immunol. 103: 119-124.

3. Role of the Nh (Non-hair) Mutation in the Development of Dermatitis and Hyperproduction of IgE in DS-Nh Mice

4. Characterization of dermatitis arising spontaneously in DS-Nh mice maintained under conventional conditions: another possible model for atopic dermatitis

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3. Scutellarein attenuates atopic dermatitis by selectively inhibiting transient receptor potential vanilloid 3 channels;British Journal of Pharmacology;2022-07-21

4. Enhanced Thermal Sensitivity of TRPV3 in Keratinocytes Underlies Heat-Induced Pruritogen Release and Pruritus in Atopic Dermatitis;Journal of Investigative Dermatology;2020-11

5. TRPV3 enhances skin keratinocyte proliferation through EGFR-dependent signaling pathways;Cell Biology and Toxicology;2020-06-13

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