Cell immortalization facilitates prelamin A clearance by increasing both cell proliferation and autophagic flux-Reference-Cited by-同舟云学术

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Cell immortalization facilitates prelamin A clearance by increasing both cell proliferation and autophagic flux

Published:2022-03-08 Issue:5 Volume:14 Page:2047-2061
ISSN:1945-4589
Container-title:Aging
language:en
Short-container-title:Aging

Author:

González-Blanco Carlos¹,Marqués Patricia¹,Burillo Jesús¹²,Jiménez Beatriz¹,García Gema¹,Benito Manuel¹³²,Guillén Carlos¹³²

Affiliation:

1. Department of Biochemistry, Complutense University, Madrid, Spain

2. Mechanisms of Insulin Resistance (MOIR2), General Direction of Universities and Investigation (CCMM), Madrid, Spain

3. Centro de Investigación Biomédica en Red (CIBER) de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM), Madrid, Spain

Publisher

Impact Journals, LLC

Subject

Cell Biology,Aging

Reference35 articles.

1. Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome.;Durkin;Nature,2003

2. Genomic instability in laminopathy-based premature aging.;Cadiñanos;Nat Med,2005

3. Lamin A and ZMPSTE24 (FACE-1) defects cause nuclear disorganization and identify restrictive dermopathy as a lethal neonatal laminopathy.;Van Essen;Hum Mol Genet,2004

4. Inhibiting farnesylation of progerin prevents the characteristic nuclear blebbing of Hutchinson-Gilford progeria syndrome.;Collins;Proc Natl Acad Sci U S A,2005

5. Blocking protein farnesyltransferase improves nuclear blebbing in mouse fibroblasts with a targeted Hutchinson-Gilford progeria syndrome mutation.;Fong;Proc Natl Acad Sci U S A,2005

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