Myeloid-specific knockout of Notch-1 inhibits MyD88- and TRIF-mediated TLR signaling pathways by regulating oxidative stress-SHP2 axis, thus restraining aneurysm progression-Reference-Cited by-同舟云学术

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Myeloid-specific knockout of Notch-1 inhibits MyD88- and TRIF-mediated TLR signaling pathways by regulating oxidative stress-SHP2 axis, thus restraining aneurysm progression

Published:2024-01-26 Issue:2 Volume:16 Page:1182-1191
ISSN:1945-4589
Container-title:Aging
language:en
Short-container-title:Aging

Author:

Li Yu¹,Guo Ailin¹,Liu Jianlei¹,Tang Lijuan²,Su Lide¹,Liu Zonghong¹

Affiliation:

1. Department of Cardiovascular Surgery, Xiang’an Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen 361102, China

2. Institute of Prevention and Control of Non-communicable Chronic Diseases, Hebei Province Center for Disease Prevention and Control, Shijiazhuang 050021, China

Publisher

Impact Journals, LLC

Reference27 articles.

1. Galectin-3 Modulates Macrophage Activation and Contributes Smooth Muscle Cells Apoptosis in Abdominal Aortic Aneurysm Pathogenesis.;Shih;Int J Mol Sci,2020

2. Decreased vascular smooth muscle cell density in medial degeneration of human abdominal aortic aneurysms.;Thompson;Am J Pathol,1997

3. Role of oxidative stress in the pathogenesis of abdominal aortic aneurysms.;Weintraub;Arterioscler Thromb Vasc Biol,2007

4. Inflammatory Mechanisms of Cardiovascular Remodeling.;Anzai;Circ J,2018

5. Combining detection of Notch1 and tumor necrosis factor-α converting enzyme is a reliable biomarker for the diagnosis of abdominal aortic aneurysms.;Zheng;Life Sci,2015

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