Dectin-1 stimulation promotes a distinct inflammatory signature in the setting of HIV-infection and aging-Reference-Cited by-同舟云学术

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Dectin-1 stimulation promotes a distinct inflammatory signature in the setting of HIV-infection and aging

Published:2023-08-21 Issue:16 Volume:15 Page:7866-7908
ISSN:1945-4589
Container-title:Aging
language:en
Short-container-title:Aging

Author:

Kumar Archit¹,Wang Jiawei²,Esterly Allen¹,Radcliffe Chris¹,Zhou Haowen²,Wyk Brent Vander³,Allore Heather G.³,Tsang Sui³,Barakat Lydia⁴,Mohanty Subhasis¹,Zhao Hongyu²,Shaw Albert C.¹,Zapata Heidi J.¹

Affiliation:

1. Yale School of Medicine, Section of Infectious Diseases, Department of Internal Medicine, New Haven, CT 06520-8022, USA

2. Interdepartmental Program in Computational Biology and Bioinformatics, Yale University, New Haven, CT 06520-8022, USA

3. Yale University Program on Aging, Yale University, New Haven, CT 06520-8022, USA

4. Yale University, Yale AIDS Care Program, New Haven, CT 06520-8022, USA

Publisher

Impact Journals, LLC

Subject

Cell Biology,Aging

Reference75 articles.

1. C-type lectin-like receptors of the dectin-1 cluster: ligands and signaling pathways.;Brown;Int Rev Immunol,2013

2. The role of Dectin-1 in the host defence against fungal infections.;Brown;Curr Opin Microbiol,2011

3. Dectin-1 is required for beta-glucan recognition and control of fungal infection.;Brown;Nat Immunol,2007

4. Identification of a human homologue of the dendritic cell-associated C-type lectin-1, dectin-1.;Ariizumi;Gene,2001

5. Dectin-1 is expressed in human lung and mediates the proinflammatory immune response to nontypeable Haemophilus influenzae.;Slevogt;mBio,2014

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