Lipocalin 2 over-expression facilitates progress of castration-resistant prostate cancer via improving androgen receptor transcriptional activity
Author:
Affiliation:
1. Department of Urology, Huashan Hospital, Fudan University, Shanghai, China
2. Department of Urology, Wake Forest University School of Medicine, Winston-Salem, North Carolina, USA
Publisher
Impact Journals, LLC
Subject
Oncology
Cited by 19 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Emerging proteins involved in castration‑resistant prostate cancer via the AR‑dependent and AR‑independent pathways (Review);International Journal of Oncology;2023-09-20
2. Lipocalin-2 regulates the expression of interferon-stimulated genes and the susceptibility of prostate cancer cells to oncolytic virus infection;European Journal of Cell Biology;2023-06
3. Thromboinflammatory Processes at the Nexus of Metabolic Dysfunction and Prostate Cancer: The Emerging Role of Periprostatic Adipose Tissue;Cancers;2022-03-25
4. Proteomic Analysis of Intracellular and Membrane-Associated Fractions of Canine (Canis lupus familiaris) Epididymal Spermatozoa and Sperm Structure Separation;Animals;2022-03-18
5. Lipocalin-2 (LCN2) Deficiency Leads to Cellular Changes in Highly Metastatic Human Prostate Cancer Cell Line PC-3;Cells;2022-01-13
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