Mechanisms for mTORC1 activation and synergistic induction of apoptosis by ruxolitinib and BH3 mimetics or autophagy inhibitors in JAK2-V617F-expressing leukemic cells including newly established PVTL-2
Author:
Affiliation:
1. Department of Hematology, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University, Tokyo, Japan
Publisher
Impact Journals, LLC
Subject
Oncology
Cited by 19 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. BH3 Mimetic Peptides: An Effective Strategy to Complement Anticancer Therapy;Current Protein & Peptide Science;2023-12
2. Vacuolar ATPase Is a Possible Therapeutic Target in Acute Myeloid Leukemia: Focus on Patient Heterogeneity and Treatment Toxicity;Journal of Clinical Medicine;2023-08-25
3. Targeting PP2A-dependent autophagy enhances sensitivity to ruxolitinib in JAK2V617F myeloproliferative neoplasms;Blood Cancer Journal;2023-07-10
4. Autophagy and SARS-CoV-2-Old Players in New Games;International Journal of Molecular Sciences;2023-04-23
5. Combined treatment with ruxolitinib and MK-2206 inhibits the JAK2/STAT5 and PI3K/AKT pathways via apoptosis in MDA-MB-231 breast cancer cell line;Molecular Biology Reports;2022-11-04
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