EglN2 contributes to triple negative breast tumorigenesis by functioning as a substrate for the FBW7 tumor suppressor-Reference-Cited by-同舟云学术

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EglN2 contributes to triple negative breast tumorigenesis by functioning as a substrate for the FBW7 tumor suppressor

Published:2016-12-27 Issue:4 Volume:8 Page:6787-6795
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Takada Mamoru¹,Zhuang Ming²,Inuzuka Hiroyuki³,Zhang Jing¹,Zurlo Giada¹,Zhang Jinfang³,Zhang Qing¹⁴

Affiliation:

1. Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA

2. Department of General Surgery, Xinhua Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, China

3. Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02215, USA

4. Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, NC 27599, USA

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 17 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. miR-161 inhibits ferroptosis by targeting EGLN2 in vascular dementia rat model;2024-08-16

2. PHD1-3 oxygen sensors in vivo—lessons learned from gene deletions;Pflügers Archiv - European Journal of Physiology;2024-03-21

3. Fbxw7 suppresses carcinogenesis and stemness in triple-negative breast cancer through CHD4 degradation and Wnt/β-catenin pathway inhibition;Journal of Translational Medicine;2024-01-24

4. FBXW7 in breast cancer: mechanism of action and therapeutic potential;Journal of Experimental & Clinical Cancer Research;2023-09-02

5. The ubiquitin–proteasome system in breast cancer;Trends in Molecular Medicine;2023-08

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