Oncogenic driver FGFR3-TACC3 requires five coiled-coil heptads for activation and disulfide bond formation for stability-Reference-Cited by-同舟云学术

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Oncogenic driver FGFR3-TACC3 requires five coiled-coil heptads for activation and disulfide bond formation for stability

Published:2023-02-11 Issue:1 Volume:14 Page:133-145
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Wang Clark G.¹²,Peiris Malalage N.¹,Meyer April N.¹,Nelson Katelyn N.¹,Donoghue Daniel J.¹³

Affiliation:

1. Department of Chemistry and Biochemistry, University of California San Diego, La Jolla, CA 92093, USA

2. Biomedical Sciences Graduate Program, University of California San Diego, La Jolla, CA 92093, USA

3. Moores UCSD Cancer Center, University of California San Diego, La Jolla, CA 92093, USA

Publisher

Impact Journals, LLC

Subject

Oncology

Reference33 articles.

1. Functions of Fibroblast Growth Factor Receptors in cancer defined by novel translocations and mutations;Donoghue;Cytokine Growth Factor Rev,2015

2. BCR: a promiscuous fusion partner in hematopoietic disorders;Donoghue;Oncotarget,2019

3. Oncogenic Gene Fusion FGFR3-TACC3 Is Regulated by Tyrosine Phosphorylation;Donoghue;Mol Cancer Res,2016

4. Oncogenic fusion protein FGFR2-PPHLN1: Requirements for biological activation, and efficacy of inhibitors;Donoghue;Transl Oncol,2020

5. Transforming fusions of FGFR and TACC genes in human glioblastoma;Ceccarelli;Science,2012

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1. Decoding Oncofusions: Unveiling Mechanisms, Clinical Impact, and Prospects for Personalized Cancer Therapies;Cancers;2023-07-19

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