Retrograde TrkAIII transport from ERGIC to ER: a re-localisation mechanism for oncogenic activity
Author:
Affiliation:
1. Department of Applied Clinical and Biotechnological Sciences, University of L'Aquila, L'Aquila, Italy
2. Department of Medical-Surgical Science and Biotechnology, University of Rome “La Sapienza”, Latina, Italy
Publisher
Impact Journals, LLC
Subject
Oncology
Cited by 17 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Molecular Characterization and Inhibition of a Novel Stress-Induced Mitochondrial Protecting Role for Misfolded TrkAIII in Human SH-SY5Y Neuroblastoma Cells;International Journal of Molecular Sciences;2024-05-17
2. A Study of Alternative TrkA Splicing Identifies TrkAIII as a Novel Potentially Targetable Participant in PitNET Progression;Biology;2024-03-07
3. Golgi retention and oncogenic KIT signaling via PLCγ2-PKD2-PI4KIIIβ activation in gastrointestinal stromal tumor cells;Cell Reports;2023-09
4. The Alternative TrkAIII Splice Variant, a Targetable Oncogenic Participant in Human Cutaneous Malignant Melanoma;Cells;2023-01-05
5. Golgi retention and oncogenic KIT signaling via PLCγ2-PKD2-PI4KIIIβ activation in GIST cells;2022-12-20
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