Selective JAK2/ABL dual inhibition therapy effectively eliminates TKI-insensitive CML stem/progenitor cells-Reference-Cited by-同舟云学术

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Selective JAK2/ABL dual inhibition therapy effectively eliminates TKI-insensitive CML stem/progenitor cells

Published:2014-08-16 Issue:18 Volume:5 Page:8637-8650
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Lin Hanyang¹²,Chen Min¹,Rothe Katharina¹³,Lorenzi Matthew V.⁴,Woolfson Adrian⁴,Jiang Xiaoyan¹²³

Affiliation:

1. Terry Fox Laboratory, British Columbia Cancer Agency, University of British Columbia; Vancouver, BC, Canada V5Z 1L3

2. Department of Medicine, University of British Columbia, University of British Columbia; Vancouver, BC, Canada V5Z 1L3

3. Department of Medical Genetics, University of British Columbia; Vancouver, BC, Canada V5Z 1L3

4. Discovery Medicine Oncology, Bristol-Myers Squibb, Princeton, NJ, United States

Publisher

Impact Journals, LLC

Subject

Oncology

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1. Targeting Phosphatases and Kinases: How to Checkmate Cancer;Frontiers in Cell and Developmental Biology;2021-10-28

2. Feeder-free and serum-free in vitro assay for measuring the effect of drugs on acute and chronic myeloid leukemia stem/progenitor cells;Experimental Hematology;2020-10

3. DYRK2 controls a key regulatory network in chronic myeloid leukemia stem cells;Experimental & Molecular Medicine;2020-10

4. Integrin-Linked Kinase Mediates Therapeutic Resistance of Quiescent CML Stem Cells to Tyrosine Kinase Inhibitors;Cell Stem Cell;2020-07

5. AMPK activation induced by promethazine increases NOXA expression and Beclin-1 phosphorylation and drives autophagy-associated apoptosis in chronic myeloid leukemia;Chemico-Biological Interactions;2020-01

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