A novel curcumin analog inhibits canonical and non-canonical functions of telomerase through STAT3 and NF-κB inactivation in colorectal cancer cells-Reference-Cited by-同舟云学术

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A novel curcumin analog inhibits canonical and non-canonical functions of telomerase through STAT3 and NF-κB inactivation in colorectal cancer cells

Published:2019-07-16 Issue:44 Volume:10 Page:4516-4531
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Chung Seyung S.¹²,Dutta Pranabananda¹,Chard Nathaniel¹,Wu Yong¹²,Chen Qiao-Hong³,Chen Guanglin³,Vadgama Jaydutt¹⁴²

Affiliation:

1. Division of Cancer Research and Training, Charles R. Drew University of Medicine and Science, Los Angeles, California 90059, USA

2. David Geffen School of Medicine, UCLA, Los Angeles, California 90095, USA

3. Department of Chemistry, California State University at Fresno, Fresno, California 93740, USA

4. Jonsson Comprehensive Cancer Center, UCLA, Los Angeles, California 90095, USA

Publisher

Impact Journals, LLC

Subject

Oncology

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2. Curcumin, its derivatives, and their nanoformulations: Revolutionizing cancer treatment;Cell Biochemistry and Function;2024-01

3. STAT3 as a newly emerging target in colorectal cancer therapy: Tumorigenesis, therapy response, and pharmacological/nanoplatform strategies;Environmental Research;2023-09

4. Research progress of small-molecule drugs in targeting telomerase in human cancer and aging;Chemico-Biological Interactions;2023-09

5. Exploring the prospective of curcumin-loaded nanomedicine in brain cancer therapy: An overview of recent updates and patented nanoformulations;Recent Patents on Nanotechnology;2023-08-23

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