UXT, a novel MDMX-binding protein, promotes glycolysis by mitigating p53-mediated restriction of NF-κB activity-Reference-Cited by-同舟云学术

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UXT, a novel MDMX-binding protein, promotes glycolysis by mitigating p53-mediated restriction of NF-κB activity

Published:2015-05-07 Issue:19 Volume:6 Page:17584-17593
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Qi Min¹,Ganapathy Suthakar²,Zeng Weiqi³,Zhang Jianglin³,Little John B.²,Yuan Zhi-Min²

Affiliation:

1. Department of plastic surgery, Xiangya Hospital, Central South University, Changsha, Hunan, PR China

2. Department of Genetics and Complex Diseases, Harvard T.H Chan School of Public Health, Boston, MA, USA

3. Department of Dermatology, Xiangya Hospital, Central South University, Changsha, Hunan, PR China

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 11 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MYB regulates the SUMO protease SENP1 and its novel interaction partner UXT, modulating MYB target genes and the SUMO landscape;Journal of Biological Chemistry;2023-09

2. UXT at the crossroads of cell death, immunity and neurodegenerative diseases;Frontiers in Oncology;2023-04-19

3. Epigenomic and somatic mutations of pituitary tumors with clinical and pathological correlations in 111 patients;Clinical Endocrinology;2022-10-07

4. The role of UXT in tumors and prospects for its application in hepatocellular carcinoma;Future Oncology;2022-09

5. UXT, a novel DNMT3b-binding protein, promotes breast cancer progression via negatively modulating lncRNA MEG3/p53 axis;Molecular Therapy - Oncolytics;2021-12

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