A new role of the Rac-GAP β2-chimaerin in cell adhesion reveals opposite functions in breast cancer initiation and tumor progression-Reference-Cited by-同舟云学术

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A new role of the Rac-GAP β2-chimaerin in cell adhesion reveals opposite functions in breast cancer initiation and tumor progression

Published:2016-04-05 Issue:19 Volume:7 Page:28301-28319
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Casado-Medrano Victoria¹,Barrio-Real Laura²,García-Rostán Ginesa¹,Baumann Matti³,Rocks Oliver³,Caloca María J.¹

Affiliation:

1. Instituto de Biología y Genética Molecular (IBGM), Consejo Superior de Investigaciones Científicas (CSIC), Universidad de Valladolid, 47003 Valladolid, Spain

2. Current address: Department of Pharmacology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104, USA

3. Max Delbrück Center for Molecular Medicine, 13125 Berlin, Germany

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 12 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fixing the GAP: The role of RhoGAPs in cancer;European Journal of Cell Biology;2022-04

2. The emerging roles of srGAPs in cancer;Molecular Biology Reports;2021-11-25

3. Mechanistic Differences of Activation of Rac1P29S and Rac1A159V;The Journal of Physical Chemistry B;2021-04-13

4. Identification of a truncated β1-chimaerin variant that inactivates nuclear Rac1;Journal of Biological Chemistry;2020-01

5. Identification of a truncated β1-chimaerin variant that inactivates nuclear Rac1;Journal of Biological Chemistry;2019-12-22

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