Mitochondrial reactive oxygen species-mediated NLRP3 inflammasome activation contributes to aldosterone-induced renal tubular cells injury-Reference-Cited by-同舟云学术

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Mitochondrial reactive oxygen species-mediated NLRP3 inflammasome activation contributes to aldosterone-induced renal tubular cells injury

Published:2016-03-21 Issue:14 Volume:7 Page:17479-17491
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Ding Wei¹,Guo Honglei²,Xu Chengyan³,Wang Bin³,Zhang Minmin³,Ding Feng¹

Affiliation:

1. Division of Nephrology, Shanghai Ninth People's Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, China

2. Division of Nephrology, The Fifth People's Hospital of Shanghai, Fudan University, Shanghai, China

3. Division of Nephrology, Huashan Hospital and Institute of Nephrology, Fudan University, Shanghai, China

Publisher

Impact Journals, LLC

Subject

Oncology

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1. Activation of the Nrf2/ARE signaling pathway ameliorates hyperlipidemia-induced renal tubular epithelial cell injury by inhibiting mtROS-mediated NLRP3 inflammasome activation;Frontiers in Immunology;2024-04-24

2. Effects of the mineralocorticoid receptor antagonist eplerenone in experimental autoimmune encephalomyelitis;The Journal of Steroid Biochemistry and Molecular Biology;2024-04

3. Aldosterone and Aldosterone Synthase Inhibitors in Cardiorenal Disease;American Journal of Physiology-Heart and Circulatory Physiology;2023-12-22

4. Design and discovery of a highly potent ultralong-acting GLP-1 and glucagon co-agonist for attenuating renal fibrosis;Acta Pharmaceutica Sinica B;2023-11

5. Mitochondrial antiviral signaling protein: a potential therapeutic target in renal disease;Frontiers in Immunology;2023-10-12

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