PD2/Paf1 depletion in pancreatic acinar cells promotes acinar-to-ductal metaplasia-Reference-Cited by-同舟云学术

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PD2/Paf1 depletion in pancreatic acinar cells promotes acinar-to-ductal metaplasia

Published:2014-05-30 Issue:12 Volume:5 Page:4480-4491
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Dey Parama¹,Rachagani Satyanarayana¹,Vaz Arokia P.¹,Ponnusamy Moorthy P.¹,Batra Surinder K.¹²³

Affiliation:

1. Department of Biochemistry and Molecular Biology, University of Nebraska Medical Center, Omaha, NE, U.S.A

2. Fred & Pamela Buffet Cancer Center, Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, NE, U.S.A

3. Department of Pathology and Microbiology, University of Nebraska Medical Center, Omaha, NE, U.S.A

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. PAF1/HIF1α axis rewires the glycolytic metabolism to fuel aggressiveness of pancreatic cancer;Cancer & Metabolism;2024-09-06

2. Elevated PAF1-RAD52 axis confers chemoresistance to human cancers;Cell Reports;2023-02

3. Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis;Oncogene;2023-01-09

4. PAF1 cooperates with YAP1 in metaplastic ducts to promote pancreatic cancer;Cell Death & Disease;2022-10-01

5. Clinical significance of pancreatic ductal metaplasia;The Journal of Pathology;2022-03-16

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