Disruption of STAT3-DNMT1 interaction by SH-I-14 induces re-expression of tumor suppressor genes and inhibits growth of triple-negative breast tumor-Reference-Cited by-同舟云学术

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Disruption of STAT3-DNMT1 interaction by SH-I-14 induces re-expression of tumor suppressor genes and inhibits growth of triple-negative breast tumor

Published:2015-05-09 Issue:48 Volume:8 Page:83457-83468
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Kang Hyo Jin¹,Yi Yong Weon¹,Hou Shu-Jie¹²,Kim Hee Jeong¹,Kong Yali¹²,Bae Insoo¹³²⁴,Brown Milton L.¹²⁴

Affiliation:

1. Department of Oncology, Georgetown University Medical Center, Washington, DC, USA

2. Center for Drug Discovery, Georgetown University Medical Center, Washington, DC, USA

3. Department of Radiation Medicine, Georgetown University Medical Center, Washington, DC, USA

4. Lombardi Comprehensive Cancer Center, Georgetown University Medical Center, Washington, DC, USA

Publisher

Impact Journals, LLC

Subject

Oncology

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2. Asymmetric post-translational modifications regulate the nuclear translocation of STAT3 homodimers in response to leukemia inhibitory factor;Cellular Oncology;2023-12-27

3. A review of biological targets and therapeutic approaches in the management of triple-negative breast cancer;Journal of Advanced Research;2023-12

4. The role of understudied post‐translational modifications for the behavior and function of Signal Transducer and Activator of Transcription 3;The FEBS Journal;2021-07-17

5. Carbazole Derivatives as STAT Inhibitors: An Overview;Applied Sciences;2021-07-03

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