Increased amyloid β-peptide uptake in skeletal muscle is induced by hyposialylation and may account for apoptosis in GNE myopathy-Reference-Cited by-同舟云学术

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Increased amyloid β-peptide uptake in skeletal muscle is induced by hyposialylation and may account for apoptosis in GNE myopathy

Published:2016-03-08 Issue:12 Volume:7 Page:13354-13371
ISSN:1949-2553
Container-title:Oncotarget
language:en
Short-container-title:Oncotarget

Author:

Bosch-Morató Mònica¹,Iriondo Cinta¹,Guivernau Biuse¹,Valls-Comamala Victòria¹,Vidal Noemí²,Olivé Montse²,Querfurth Henry³,Muñoz Francisco J.¹

Affiliation:

1. Laboratory of Molecular Physiology, Department of Experimental and Health Sciences, Universitat Pompeu Fabra, Barcelona, Spain

2. Institut de Neuropatologia, Servei Anatomia Patològica, Hospital de Bellvitge, Hospitalet de Llobregat, Barcelona, Spain

3. Department of Neurology, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, RI, USA

Publisher

Impact Journals, LLC

Subject

Oncology

Cited by 17 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Potential small effector molecules restoring cellular defects due to sialic acid biosynthetic enzyme deficiency: Pathological relevance to GNE myopathy;Biochemical Pharmacology;2024-05

2. Understanding pathophysiology of GNE myopathy and current progress towards drug development;Journal of Biosciences;2024-02-19

3. GNE myopathy: History, etiology, and treatment trials;Frontiers in Neurology;2022-10-18

4. Role of HSP70 chaperone in protein aggregate phenomenon of GNE mutant cells: Therapeutic lead for GNE Myopathy;The International Journal of Biochemistry & Cell Biology;2022-08

5. The role of amyloid β in the pathological mechanism of GNE myopathy;Neurological Sciences;2022-07-29

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