Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation

Author:

Chien Yueh12,Chien Chian-Shiu12,Chiang Huai-Chih12,Huang Wei-Lin13,Chou Shih-Jie12,Chang Wei-Chao4,Chang Yuh-Lih52,Leu Hsin-Bang67,Chen Kuan-Hsuan57,Wang Kang-Ling67,Lai Ying-Hsiu7,Liu Yung-Yang17,Lu Kai-Hsi8,Li Hsin-Yang13,Sung Yen-Jen3,Jong Yuh-Jyh9,Chen Yann-Jang10,Chen Chung-Hsuan1112,Yu Wen-Chung613

Affiliation:

1. Department of Medical Research, Taipei Veterans General Hospital, Taipei, Taiwan

2. Institute of Pharmacology, Taipei, Taiwan

3. Institute of Anatomy and Cell Biology, Taipei, Taiwan

4. Graduate Institute of Cancer Biology and Center for Molecular Medicine, China Medical University and Department of Biotechnology, Asia University, Taichung, Taiwan

5. Department of Pharmacy, Taipei Veterans General Hospital, Taipei, Taiwan

6. Division of Cardiology & Department of Medicine, Taipei Veterans General Hospital, Taipei, Taiwan

7. Institute of Clinical Medicine, Taipei, Taiwan

8. Department of Medical Research, Cheng-Hsin Hospital, Taipei, Taiwan

9. College of Biological Science and Technology, National Chiao Tung University, Hsinchu, Taiwan

10. Department of Life Sciences and Institute of Genome Sciences, Taipei, Taiwan

11. Genomics Research Center, Academia Sinica, Taipei, Taiwan

12. Department of Chemistry, National Taiwan University, Taipei, Taiwan

13. Faculty of Medicine, National Yang-Ming University, Taipei, Taiwan

Publisher

Impact Journals, LLC

Subject

Oncology

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