Genomic deletion of GIT2 induces a premature age-related thymic dysfunction and systemic immune system disruption

Author:

Siddiqui Sana1,Lustig Ana2,Carter Arnell2,Sankar Mathavi3,Daimon Caitlin M.3,Premont Richard T.4,Etienne Harmonie5,van Gastel Jaana5,Azmi Abdelkrim5,Janssens Jonathan5,Becker Kevin G.6,Zhang Yongqing6,Wood William6,Lehrmann Elin6,Martin James G.7,Martin Bronwen3,Taub Dennis D.2,Maudsley Stuart15

Affiliation:

1. Receptor Pharmacology Unit, Laboratory of Neurosciences, National Institute on Aging (NIA), National Institutes of Health (NIH), Baltimore, MD 21224, USA

2. Laboratory of Molecular Biology and Immunology, NIA, NIH, Baltimore, MD 21224, USA

3. Metabolism Unit, Laboratory of Clinical Investigation, NIA, NIH, Baltimore, MD 21224, USA

4. Duke University Medical Center, Durham, NC 27705, USA

5. Translational Neurobiology Group, VIB Department of Molecular Genetics, University of Antwerp, Belgium

6. Gene Expression and Genomics Unit, Research Resources Branch, NIA, NIH, Baltimore, MD 21224, USA

7. Research Institute of the MUHC, Centre for Translational Biology (CTB), Meakins-Christie Laboratories, McGill University, Montreal, QC, H4A 3J1, Canada

Publisher

Impact Journals, LLC

Subject

Cell Biology,Aging

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