Disruption of mechanical stress in extracellular matrix is related to Stanford type A aortic dissection through down-regulation of Yes-associated protein-Reference-Cited by-同舟云学术

Disruption of mechanical stress in extracellular matrix is related to Stanford type A aortic dissection through down-regulation of Yes-associated protein

Published:2016-09-05 Issue:9 Volume:8 Page:1923-1939
ISSN:1945-4589
Container-title:Aging
language:en
Short-container-title:Aging

Author:

Jiang Wen-Jian¹²³⁴⁵,Ren Wei-Hong²³,Liu Xu-Jie²³,Liu Yan²³,Wu Fu-Jian²³,Sun Li-Zhong¹³⁴⁵⁶,Lan Feng²³,Du Jie²³,Zhang Hong-Jia¹²³⁴⁵

Affiliation:

1. Department of Cardiac Surgery, Beijing Anzhen Hospital, Capital Medical University, Beijing 10029, China

2. Beijing Institute of Heart, Lung and Blood Vessel Diseases, Beijing 10029, China

3. Beijing Laboratory for Cardiovascular Precision Medicine, Beijing 10029, China

4. The Key Laboratory of Remodeling-related Cardiovascular Disease, Ministry of Education, Beijing 10029, China

5. Beijing Aortic Disease Center, Cardiovascular Surgery Center, Beijing 10029, China

6. Beijing Engineering Research Center for Vascular Prostheses, Beijing, China, 10029

Publisher

Impact Journals, LLC

Subject

Cell Biology,Aging

Cited by 22 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Downregulation of RhoA/ROCK1/YAP/F-actin causing decreased aortic smooth muscle cell stiffness promotes aortic dissection formation;Life Metabolism;2024-06-03

2. Unveiling cellular and molecular aspects of ascending thoracic aortic aneurysms and dissections;Basic Research in Cardiology;2024-05-03

3. Dysregulated Genes and Signaling Pathways in the Formation and Rupture of Intracranial Aneurysm;Translational Stroke Research;2023-08-30

4. Mammalian sterile 20-like kinase 1 acts as a candidate biomarker of mortality of emergency surgical repair for acute type a aortic dissection;BMC Cardiovascular Disorders;2023-04-10

5. Yes-Associated Protein and Transcriptional Coactivator with PDZ-Binding Motif in Cardiovascular Diseases;International Journal of Molecular Sciences;2023-01-14