Modulation of mitochondrial adenosine triphosphate-sensitive potassium channels and sodium-hydrogen exchange provide additive protection from severe ischemia-reperfusion injury-Reference-Cited by-同舟云学术

Modulation of mitochondrial adenosine triphosphate-sensitive potassium channels and sodium-hydrogen exchange provide additive protection from severe ischemia-reperfusion injury

Published:2003-04 Issue:4 Volume:125 Page:863-871
ISSN:0022-5223
Container-title:The Journal of Thoracic and Cardiovascular Surgery
language:en
Short-container-title:The Journal of Thoracic and Cardiovascular Surgery

Author:

Digerness Stanley B.,Brookes Paul S.,Goldberg Steven P.,Katholi Charles R.,Holman William L.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Pulmonary and Respiratory Medicine,Surgery

Reference34 articles.

1. Ischemic preconditioning with opening of mitochondrial adenosine triphosphate-sensitive potassium channels or Na+/H+ exchange inhibition: which is the best protective strategy for heart transplants?;Kevelaitis;J Thorac Cardiovasc Surg,2001

2. Roles of mitochondrial ATP-sensitive K channels and PKC in anti-infarct tolerance afforded by adenosine A1 receptor activation;Miura;J Am Coll Cardiol,2000

3. Opening of mitochondrial KATP channels triggers the preconditioned state by generating free radicals;Pain;Circ Res,2000

4. Increased mitochondrial KATP channel activity during chronic myocardial hypoxia: is cardioprotection mediated by improved bioenergetics?;Eells;Circ Res,2000

5. Activation of mitochondrial K+ATP channel elicits late preconditioning against myocardial infarction via protein kinase C signaling pathway;Takashi;Circ Res,1999

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1. Myocardial stunning following no flow ischaemia is diminished by levosimendan or cariporide, without benefits of combined administration;Resuscitation;2008-01

2. Cardioprotection and mitochondrial S-nitrosation: Effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia–reperfusion injury;Journal of Molecular and Cellular Cardiology;2007-04

3. Sodium–Hydrogen Exchange Inhibition and β-Blockade Additively Decrease Infarct Size;The Annals of Thoracic Surgery;2007-03

4. Mitochondrial dysfunction in cardiac ischemia–reperfusion injury: ROS from complex I, without inhibition;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2006-02

5. Multiple Treatment Approach to Limit Cardiac Ischemia-Reperfusion Injury;The Annals of Thoracic Surgery;2005-10