Anchored PDE4 regulates chloride conductance in wild‐type and ΔF508‐CFTR human airway epithelia-Reference-Cited by-同舟云学术

Anchored PDE4 regulates chloride conductance in wild‐type and ΔF508‐CFTR human airway epithelia

Published:2013-11-07 Issue:2 Volume:28 Page:791-801
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Blanchard Elise¹,Zlock Lorna²,Lao Anna²,Mika Delphine¹,Namkung Wan³⁴⁵,Xie Moses¹,Scheitrum Colleen¹,Gruenert Dieter C.⁶,Verkman Alan S.³⁴,Finkbeiner Walter E.²,Conti Marco¹,Richter Wito¹

Affiliation:

1. Department of Obstetrics, Gynecology, and Reproductive SciencesUniversity of California San FranciscoSan FranciscoCaliforniaUSA

2. Department of PathologyUniversity of California San FranciscoSan FranciscoCaliforniaUSA

3. Department of MedicineUniversity of California San FranciscoSan FranciscoCaliforniaUSA

4. Department of PhysiologyUniversity of California San FranciscoSan FranciscoCaliforniaUSA

5. College of PharmacyYonsei Institute of Pharmaceutical SciencesYonsei UniversityIncheonKorea

6. Department of Otolaryngology‐Head and Neck SurgeryInstitute for Human GeneticsEli and Edythe Broad Center for Regenerative Medicine and Stem Cell ResearchUniversity of California San FranciscoSan FranciscoCaliforniaUSA

Funder

Cystic Fibrosis Foundation

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-240861

Reference65 articles.

1. Chloride channels as drug targets

2. Insights into the mechanisms underlying CFTR channel activity, the molecular basis for cystic fibrosis and strategies for therapy