Exploiting functional domains of GRK2/3 to alter the competitive balance of pro‐ and anticontractile signaling in airway smooth muscle-Reference-Cited by-同舟云学术

Exploiting functional domains of GRK2/3 to alter the competitive balance of pro‐ and anticontractile signaling in airway smooth muscle

Published:2013-10-16 Issue:2 Volume:28 Page:956-965
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Deshpande Deepak A.¹,Yan Huandong¹,Kong Kok‐Choi²,Tiegs Brian C.³,Morgan Sarah J.¹,Pera Tonio³,Panettieri Reynold A.⁴,Eckhart Andrea D.³,Penn Raymond B.¹²³

Affiliation:

1. Department of MedicineDivision of Pulmonary and Critical Care MedicineUniversity of Maryland School of MedicineBaltimoreMarylandUSA

2. Department of Internal MedicineWake Forest University Health SciencesWinston‐SalemNorth CarolinaUSA

3. Department of MedicineCenter for Translational MedicineJane and Leonard Korman Lung CenterThomas Jefferson UniversityPhiladelphiaPennsylvaniaUSA

4. Department of MedicineUniversity of Pennsylvania School of MedicinePhiladelphiaPennsylvaniaUSA

Funder

National Institutes of Health

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-240226

Reference44 articles.

1. Targeting G protein-coupled receptor signaling in asthma

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3. β-Arrestin acts as a clathrin adaptor in endocytosis of the β2-adrenergic receptor

4. Regulation of G Protein-Coupled Receptor Kinases

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