Prohibitin 2 represents a novel nuclear AKT substrate during all‐ trans retinoic acid–induced differentiation of acute promyelocytic leukemia cells-Reference-Cited by-同舟云学术

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Prohibitin 2 represents a novel nuclear AKT substrate during all‐ trans retinoic acid–induced differentiation of acute promyelocytic leukemia cells

Published:2014-02-12 Issue:5 Volume:28 Page:2009-2019
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Bavelloni Alberto¹²,Piazzi Manuela³,Faenza Irene³,Raffini Mirco²,D'Angelo Antonietta²,Cattini Luca²,Cocco Lucio³,Blalock William L.¹⁴

Affiliation:

1. Struttura Complessa (SC) Laboratory of Musculoskeletal Cell BiologyRizzoli Orthopedic InstituteBolognaItaly

2. Ramses LaboratoryRizzoli Orthopedic InstituteBolognaItaly

3. Cell Signaling LaboratoryDepartment of Biomedical SciencesUniversity of BolognaBolognaItaly

4. National Research Council (CNR) of ItalyInstitute of Molecular Genetics (IGM)BolognaItaly

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.13-244368

Reference62 articles.

1. Partial deletion of long arm of chromosome 17: a specific abnormality in acute promyelocytic leukemia?;Golomb H. M.;Arch. Intern. Med.,1976

2. How I treat acute promyelocytic leukemia

3. Retinoid target genes in acute promyelocytic leukemia

4. Retinoic acid receptors, hematopoiesis and leukemogenesis

5. Eradication of acute promyelocytic leukemia-initiating cells through PML-RARA degradation

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