Intronic polyadenylation signal sequences and alternate splicing generate human soluble Fltl variants and regulate the abundance of soluble Flt1 in the placenta-Reference-Cited by-同舟云学术

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Intronic polyadenylation signal sequences and alternate splicing generate human soluble Fltl variants and regulate the abundance of soluble Flt1 in the placenta

Published:2007-07-05 Issue:14 Volume:21 Page:3885-3895
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Thomas Christie P.¹²,Andrews Janet I.³,Liu Kang Z.¹

Affiliation:

1. Departments of Internal MedicineDepartments of Obstetrics and GynecologyUniversity of IowaDepartments of Veterans Affairs Medical CenterIowa CityIowaUSA

2. Departments of Veterans Affairs Medical CenterIowa CityIowaUSA

3. Departments of Obstetrics and GynecologyUniversity of IowaDepartments of Veterans Affairs Medical CenterIowa CityIowaUSA

Funder

American Society of Nephrology

U.S. Public Health Service

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.07-8809com

Reference50 articles.

1. The splice variants of vascular endothelial growth factor (VEGF) and their receptors;Robinson C. J.;J. Cell Sci.,2001

2. VEGF-receptor signal transduction

3. Signal transduction by VEGF receptors in regulation of angiogenesis and lymphangiogenesis

4. Inhibition of vascular endothelial cell growth factor activity by an endogenously encoded soluble receptor.

5. Genomic organization of the flt-1 gene encoding for Vascular Endothelial Growth Factor (VEGF) Receptor-1 suggests an intimate evolutionary relationship between the 7-Ig and the 5-Ig tyrosine kinase receptors

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