Are genetically modified mice useful for the understanding of acute pancreatitis?
Author:
Affiliation:
1. Division d’Hépatologie et de Gastroentérologie, Geneva, Switzerland
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Reference31 articles.
1. Rinderknecht, H. (1988) Fatal pancreatitis, a consequence of excessive leukocyte stimulation? Int. J. Pancreatol. 3,105-112
2. Acute Pancreatitis-Induced Enzyme Release and Necrosis Are Attenuated by IL-1 Antagonism through an Indirect Mechanism
3. Tissue-specific regulation of glutamine synthetase gene expression in acute pancreatitis is confirmed by using interleukin-1 receptor knockout mice
4. Transgenic Animals Demonstrate a Role for the IL-1 Receptor in Regulating IL-1β Gene Expression at Steady-State and during the Systemic Stress Induced by Acute Pancreatitis
5. Evidence for an unknown component of pancreatic ascites that induces adult respiratory distress syndrome through an interleukin-1 and tumor necrosis factor-dependent mechanism
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