Constitutive expression of arginase in microvascular endothelial cells counteracts nitric oxide‐mediated vasodilatory function-Reference-Cited by-同舟云学术

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Constitutive expression of arginase in microvascular endothelial cells counteracts nitric oxide‐mediated vasodilatory function

Published:2001-03-12 Issue:7 Volume:15 Page:1264-1266
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zhang Cuihua¹,Hein Travis W.¹,Wang Wei¹,Chang Chiung‐I¹,Kuo Lih¹

Affiliation:

1. Department of Medical Physiology Cardiovascular Research Institute, The Texas A&M University System Health Science Center College Station Texas

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.00-0681fje

Reference38 articles.

1. Arginine metabolism in experimental glomerulonephritis: interaction between nitric oxide synthase and arginase;Cook H. T.;Am. J. Physiol.,1994

2. Arginase modulates nitric oxide production in activated macrophages

3. Nitric oxide synthase isozymes. Characterization, purification, molecular cloning, and functions.

4. Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide.

5. L-arginine is the physiological precursor for the formation of nitric oxide in endothelium-dependent relaxation

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