Targeted STIM deletion impairs calcium homeostasis, NFAT activation, and growth of smooth muscle-Reference-Cited by-同舟云学术

Targeted STIM deletion impairs calcium homeostasis, NFAT activation, and growth of smooth muscle

Published:2012-11-16 Issue:3 Volume:27 Page:893-906
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Mancarella Salvatore¹²,Potireddy Santhi¹,Wang Youjun¹,Gao Hui³²,Gandhirajan Rajesh Kumar¹,Autieri Michael³²,Scalia Rosario³²,Cheng Zhongjian⁴,Wang Hong⁴,Madesh Muniswamy¹,Houser Steven R.³²,Gill Donald L.¹²

Affiliation:

1. Department of BiochemistryTemple University School of MedicinePhiladelphiaPennsylvaniaUSA

2. Cardiovascular Research CenterTemple University School of MedicinePhiladelphiaPennsylvaniaUSA

3. Department of PhysiologyTemple University School of MedicinePhiladelphiaPennsylvaniaUSA

4. Department of PharmacologyTemple University School of MedicinePhiladelphiaPennsylvaniaUSA

Funder

National Institutes of Health

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.12-215293

Reference67 articles.

1. STIM1, an essential and conserved component of store-operated Ca2+ channel function

2. STIM Is a Ca2+ Sensor Essential for Ca2+-Store-Depletion-Triggered Ca2+ Influx

3. Store-Operated Calcium Channels: New Perspectives on Mechanism and Function

4. STIM Proteins and the Endoplasmic Reticulum-Plasma Membrane Junctions

5. STIM proteins: dynamic calcium signal transducers

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