Complement‐induced activation of the cardiac NLRP3 inflammasome in sepsis-Reference-Cited by-同舟云学术

Complement‐induced activation of the cardiac NLRP3 inflammasome in sepsis

Published:2016-08-19 Issue:12 Volume:30 Page:3997-4006
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Kalbitz Miriam¹²,Fattahi Fatemeh¹,Grailer Jamison J.¹,Jajou Lawrence¹,Malan Elizabeth A.¹,Zetoune Firas S.¹,Huber‐Lang Markus²,Russell Mark W.³,Ward Peter A.¹

Affiliation:

1. Department of Pathology Ann Arbor Michigan USA

2. Department of Orthopaedic TraumaHand, Plastic, and Reconstructive Surgery, University Hospital of Ulm Ulm Germany

3. Department of Pediatric CardiologyUniversity of Michigan Medical School Ann Arbor Michigan USA

Funder

National Institutes of Health

National Institute of General Medical Sciences

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201600728R

Reference56 articles.

1. The inflammasome: a caspase-1-activation platform that regulates immune responses and disease pathogenesis

2. Inflammasomes: too big to miss

3. Role of extracellular histones in the cardiomyopathy of sepsis

4. Gout-associated uric acid crystals activate the NALP3 inflammasome

5. NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

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