Role of HSP60 (HSPD1) in diabetes‐induced renal tubular dysfunction: regulation of intracellular protein aggregation, ATP production, and oxidative stress-Reference-Cited by-同舟云学术

Role of HSP60 (HSPD1) in diabetes‐induced renal tubular dysfunction: regulation of intracellular protein aggregation, ATP production, and oxidative stress

Published:2017-02-14 Issue:5 Volume:31 Page:2157-2167
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Aluksanasuwan Siripat¹²,Sueksakit Kanyarat¹²,Fong‐ngern Kedsarin¹²,Thongboonkerd Visith¹²

Affiliation:

1. Medical Proteomics Unit, Office for Research and DevelopmentFaculty of Medicine Siriraj HospitalMahidol UniversityBangkokThailand

2. Center for Research in Complex Systems ScienceMahidol UniversityBangkokThailand

Funder

Mahidol University

Office of the Higher Education Commission

Thailand Research Fund

Faculty of Medicine Siriraj Hospital, Mahidol University

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201600910RR

Reference55 articles.

1. The tubulointerstitium in progressive diabetic kidney disease: More than an aftermath of glomerular injury?

2. High glucose inhibits glucose uptake in renal proximal tubule cells by oxidative stress and protein kinase C;Han H. J.;Kidney Int.,2000

3. Reduced albumin reabsorption in the proximal tubule of early-stage diabetic rats

4. The Role of the p38 MAPK Signaling Pathway in High Glucose-Induced Epithelial-Mesenchymal Transition of Cultured Human Renal Tubular Epithelial Cells

5. Advanced Glycation End Products Induce Tubular Epithelial-Myofibroblast Transition through the RAGE-ERK1/2 MAP Kinase Signaling Pathway

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