In situ targeting of dendritic cells sets tolerogenic environment and ameliorates CD4 + T‐cell response in the postischemic liver-Reference-Cited by-同舟云学术

In situ targeting of dendritic cells sets tolerogenic environment and ameliorates CD4 + T‐cell response in the postischemic liver

Published:2017-07-18 Issue:11 Volume:31 Page:4796-4808
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Funken Dominik¹,Ishikawa‐Ankerhold Hellen²³,Uhl Bernd²,Lerchenberger Maximilian¹,Rentsch Markus¹,Mayr Doris⁴,Massberg Steffen³,Werner Jens¹,Khandoga Andrej¹

Affiliation:

1. Department of General, Visceral, and Transplant SurgeryUniversity of Munich Munich Germany

2. Walter‐Brendel Centre of Experimental Medicine Munich Germany

3. Department of CardiologyUniversity of Munich Munich Germany

4. Department of PathologyUniversity of Munich Munich Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201601358R

Reference35 articles.

1. CD4+ T cells contribute to postischemic liver injury in mice by interacting with sinusoidal endothelium and platelets

2. CD4 T cells promote tissue inflammation via CD40 signaling without de novo activation in a murine model of liver ischemia/reperfusion injury

3. Protective Effects of Early CD4+ T Cell Reduction in Hepatic Ischemia/Reperfusion Injury

4. Liver grafts from CD39-overexpressing rodents are protected from ischemia reperfusion injury due to reduced numbers of resident CD4+T cells

5. Near-Term Anti-CD25 Monoclonal Antibody Administration Protects Murine Liver from Ischemia-Reperfusion Injury Due to Reduced Numbers of CD4+ T Cells

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