The endoplasmic reticulum remains functionally connected by vesicular transport after its fragmentation in cells expressing Z‐α 1 ‐antitrypsin-Reference-Cited by-同舟云学术

The endoplasmic reticulum remains functionally connected by vesicular transport after its fragmentation in cells expressing Z‐α 1 ‐antitrypsin

Published:2016-09-06 Issue:12 Volume:30 Page:4083-4097
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Dickens Jennifer A.¹²,Ordöñez Adriana¹,Chambers Joseph E.¹²,Beckett Alison J.³,Patel Vruti¹,Malzer Elke¹,Dominicus Caia S.¹,Bradley Jayson¹,Peden Andrew A.³,Prior Ian A.⁴,Lomas David A.⁵,Marciniak Stefan J.¹²

Affiliation:

1. Cambridge Institute for Medical Research Cambridge United Kingdom

2. Department of MedicineUniversity of Cambridge Cambridge United Kingdom

3. Department of Biomedical ScienceUniversity of Sheffield Sheffield United Kingdom

4. Institute of Translational MedicineUniversity of Liverpool Liverpool United Kingdom

5. UCL RespiratoryUniversity College London London United Kingdom

Funder

Medical Research Council

Academy of Medical Sciences

National Institute for Health Research

NIHR Biomedical Research Centre, Royal Marsden NHS Foundation Trust/Institute of Cancer Research

Alpha-1 Foundation

Wellcome Trust

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201600430R

Reference41 articles.

1. The Selective Advantage of α1-Antitrypsin Deficiency

2. Why has it been so difficult to prove the efficacy of alpha‐1‐antitrypsin replacement therapy;Dickens J. A.;Insights from the study of disease pathogenesis. Drug Des. Devel. Ther,2011

3. The mechanism of Z α1-antitrypsin accumulation in the liver

4. Risk of Cirrhosis and Primary Liver Cancer in Alpha1-Antitrypsin Deficiency

5. Pulmonary Emphysema and Alpha1-Antitrypsin Deficiency

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