Loss of laminin‐a4 results in pre‐ and postsynaptic modifications at the neuromuscular junction-Reference-Cited by-同舟云学术

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Loss of laminin‐a4 results in pre‐ and postsynaptic modifications at the neuromuscular junction

Published:2016-12-22 Issue:4 Volume:31 Page:1323-1336
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Chand Kirat K.¹,Lee Kah Meng¹,Lavidis Nickolas A.¹,Noakes Peter G.¹²

Affiliation:

1. School of Biomedical Sciences St. Lucia Queensland Australia

2. Queensland Brain InstituteThe University of Queensland St. Lucia Queensland Australia

Funder

National Health and Medical Research Council

University of Queensland

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201600899R

Reference73 articles.

1. Aberrant differentiation of neuromuscular junctions in mice lacking s-laminin/laminin β2

2. Motor neurons and Schwann cells distinguish between synaptic and extrasynaptic isoforms of laminin

3. Synaptic laminin prevents glial entry into the synaptic cleft

4. Properly formed but improperly localized synaptic specializations in the absence of laminin α4

5. Laminins promote postsynaptic maturation by an autocrine mechanism at the neuromuscular junction

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2. Aberrant evoked calcium signaling and nAChR cluster morphology in a SOD1 D90A hiPSC-derived neuromuscular model;Frontiers in Cell and Developmental Biology;2024-06-20

3. Adipocyte-Specific Laminin Alpha 4 Deletion Preserves Adipose Tissue Health despite Increasing Adiposity;Biomedicines;2022-08-25

4. Neuromuscular Junction Dysfunction in Amyotrophic Lateral Sclerosis;Molecular Neurobiology;2022-01-08

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