Hydroxysteroid (17β)-dehydrogenase 1–deficient female mice present with normal puberty onset but are severely subfertile due to a defect in luteinization and progesterone production-Reference-Cited by-同舟云学术

Hydroxysteroid (17β)-dehydrogenase 1–deficient female mice present with normal puberty onset but are severely subfertile due to a defect in luteinization and progesterone production

Published:2015-09 Issue:9 Volume:29 Page:3806-3816
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Hakkarainen Janne,Jokela Heli,Pakarinen Pirjo,Heikelä Hanna,Kätkänaho Laura,Vandenput Liesbeth,Ohlsson Claes,Zhang Fu-Ping,Poutanen Matti

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference47 articles.

1. 17beta-hydroxysteroid dehydrogenase (HSD)/17-ketosteroid reductase (KSR) family; nomenclature and main characteristics of the 17HSD/KSR enzymes

2. Structure and function of human 17β-hydroxysteroid dehydrogenases

3. SDR-type human hydroxysteroid dehydrogenases involved in steroid hormone activation

4. Multifunctionality of human 17β-hydroxysteroid dehydrogenases

5. Hydroxysteroid (17β) Dehydrogenase 7 Activity Is Essential for Fetal de Novo Cholesterol Synthesis and for Neuroectodermal Survival and Cardiovascular Differentiation in Early Mouse Embryos

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