The FK506-binding protein FKBP52in vitroinduces aggregation of truncated Tau forms with prion-like behavior-Reference-Cited by-同舟云学术

The FK506-binding protein FKBP52in vitroinduces aggregation of truncated Tau forms with prion-like behavior

Published:2015-08 Issue:8 Volume:29 Page:3171-3181
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Giustiniani Julien,Guillemeau Kevin,Dounane Omar,Sardin Elodie,Huvent Isabelle,Schmitt Alain,Hamdane Malika,Buée Luc,Landrieu Isabelle,Lippens Guy,Baulieu Etienne Emile,Chambraud Béatrice

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference46 articles.

1. Tau pathology and neurodegeneration

2. Tauopathies and Tau Oligomers

3. Tau filaments from human brain and from in vitro assembly of recombinant protein show cross- structure

4. Mechanisms of tau-induced neurodegeneration

5. The self-perpetuating tau truncation circle

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