Podocyte GSK3α is important for autophagy and its loss detrimental for glomerular function-Reference-Cited by-同舟云学术

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Podocyte GSK3α is important for autophagy and its loss detrimental for glomerular function

Published:2019-07-09 Issue:8 Volume:1 Page:498-510
ISSN:2573-9832
Container-title:FASEB BioAdvances
language:en
Short-container-title:FASEB BioAdvances

Author:

Hurcombe Jennifer A.¹,Lay Abigail C.¹,Ni Lan¹,Barrington Alexandra F.¹,Woodgett Jim R.²,Quaggin Susan E.³,Welsh Gavin I.¹,Coward Richard J.¹

Affiliation:

1. Bristol Renal University of Bristol Bristol UK

2. Lunenfeld‐Tanenbaum Research Institute Sinai Health System & University of Toronto Toronto ON Canada

3. Feinberg Cardiovascular Research Institute Northwestern University Feinberg School of Medicine Chicago Illinois

Funder

Kidney Research UK

Medical Research Council

Canadian Institutes of Health Research

National Institutes of Health

Innovative Medicines Initiative

Horizon 2020 Framework Programme

Juvenile Diabetes Research Foundation International

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fba.2019-00011

Reference45 articles.

1. The glamour and gloom of glycogen synthase kinase-3

2. Molecular cloning and expression of glycogen synthase kinase-3/factor A.

3. Spatial learning deficit in transgenic mice that conditionally over-express GSK-3β in the brain but do not form tau filaments

4. Deletion of GSK-3β in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation

5. Decreased nuclear beta-catenin, tau hyperphosphorylation and neurodegeneration in GSK-3beta conditional transgenic mice

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