Transient activation of AMPK preceding left ventricular pressure overload reduces adverse remodeling and preserves left ventricular function-Reference-Cited by-同舟云学术

Transient activation of AMPK preceding left ventricular pressure overload reduces adverse remodeling and preserves left ventricular function

Published:2018-07-19 Issue:1 Volume:33 Page:711-721
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Nam Deok Hwa¹,Kim Eunah¹,Benham Ashley²,Park Hye-Kyung¹,Soibam Benjamin³,Taffet George E.⁴,Kaelber Jason T.⁵⁶,Suh Ji Ho¹,Taegtmeyer Heinrich⁷,Entman Mark L.⁴,Reineke Erin L.¹

Affiliation:

1. Center for Bioenergetics Houston Methodist Research Institute Houston Texas USA

2. Texas Heart Institute Houston Texas USA

3. Department of Computer Science and Engineering Technology University of Houston-Downtown Houston Texas USA

4. Division of Cardiovascular Sciences Department of Medicine Baylor College of Medicine Houston Texas USA

5. National Center for Macromolecular Imaging Baylor College of Medicine Houston Texas USA

6. Department of Molecular Virology and Microbiology Baylor College of Medicine Houston Texas USA

7. Division of Cardiology Department of Internal Medicine McGovern Medical School University of Texas Health Science Center Houston Texas USA

Funder

National Institutes of Health

U.S. Department of Defense

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201800602R

Reference28 articles.

1. Steroid Receptor Coactivator-2 Is a Dual Regulator of Cardiac Transcription Factor Function

2. SRC-2 Coactivator Deficiency Decreases Functional Reserve in Response to Pressure Overload of Mouse Heart

3. AMP-Activated Protein Kinase Regulation and Biological Actions in the Heart

4. AMP-Activated Protein Kinase Regulates E3 Ligases in Rodent Heart

5. Immune-inflammatory dysregulation modulates the incidence of progressive fibrosis and diastolic stiffness in the aging heart

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