The antipyretic effect of paracetamol occurs independent of transient receptor potential ankyrin 1‐mediated hypothermia and is associated with prostaglandin inhibition in the brain-Reference-Cited by-同舟云学术

The antipyretic effect of paracetamol occurs independent of transient receptor potential ankyrin 1‐mediated hypothermia and is associated with prostaglandin inhibition in the brain

Published:2018-05-08 Issue:10 Volume:32 Page:5751-5759
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Mirrasekhian Elahe¹,Nilsson Johan L. Å.²,Shionoya Kiseko¹,Blomgren Anders²,Zygmunt Peter M.²,Engblom David¹,Högestôtt Edward D.²,Blomqvist Anders¹

Affiliation:

1. Department of Clinical and Experimental MedicineLinköping UniversityLinköpingSweden

2. Division of Clinical Chemistry and Pharmacology, Department of Laboratory MedicineLund UniversityLundSweden

Funder

Medicinska Forskningsrådet

Centre of Excellence for Core to Crust Fluid Systems, Australian Research Council

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201800272R

Reference37 articles.

1. Effects of Anti-inflammatory Drugs on Prostaglandin Biosynthesis

2. Inhibition of Prostaglandin Synthetase in Brain explains the Anti-pyretic Activity of Paracetamol (4-Acetamidophenol)

3. Mechanism of Acetaminophen Inhibition of Cyclooxygenase Isoforms

4. Determinants of the cellular specificity of acetaminophen as an inhibitor of prostaglandin H2 synthases

5. TRPA1 mediates spinal antinociception induced by acetaminophen and the cannabinoid Δ9-tetrahydrocannabiorcol

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