Fas receptor induces apoptosis of synovial bone and cartilage progenitor populations and promotes bone loss in antigen‐induced arthritis-Reference-Cited by-同舟云学术

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Fas receptor induces apoptosis of synovial bone and cartilage progenitor populations and promotes bone loss in antigen‐induced arthritis

Published:2018-11 Issue:3 Volume:33 Page:3330-3342
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Mosler Elvira Lazić¹²³,Lukač Nina¹⁴,Flegar Darja¹⁵,Fadljević Martina¹,Radanović Igor¹,Cvija Hrvoje¹,Kelava Tomislav¹⁵,Ivčević Sanja¹⁶,Šućur Alan¹⁵,Markotić Antonio¹⁷,Katavić Vedran¹⁴,Marušić Ana⁸,Grčević Danka¹⁸,Kovačić And Nataša¹⁴

Affiliation:

1. Laboratory for Molecular ImmunologyCroatian Institute for Brain ResearchUniversity of Zagreb School of Medicine Zagreb Croatia

2. Department of Dermatology and VenerologyGeneral Hospital Dr. Ivo Pedišić Sisak Croatia

3. Department of NursingCatholic University of Croatia Zagreb Croatia

4. Department of AnatomyUniversity of Zagreb School of Medicine Zagreb Croatia

5. Department of Physiology and ImmunologyUniversity of Zagreb School of Medicine Zagreb Croatia

6. Huntsman Cancer InstituteUniversity of Utah Salt Lake City Utah USA

7. Centre for Clinical PharmacologyUniversity Clinical Hospital Mostar Mostar Bosnia and Herzegovina

8. Department of Research in Biomedicine and HealthUniversity of Split School of Medicine Split Croatia

Funder

Hrvatska Zaklada za Znanost

California Institute for Regenerative Medicine

Ontario Institute for Regenerative Medicine

Institute for Translational Neuroscience

Center for Outcomes Research and Evaluation, Yale School of Medicine

European Regional Development Fund

Ministry of Science and Technology, Croatia

Ministarstvo Obrazovanja, Znanosti i Sporta

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201801426R

Reference51 articles.

1. Rheumatoid Arthritis

2. Cytokines in the pathogenesis of rheumatoid arthritis

3. Autoimmune inflammation from the Th17 perspective

4. Interleukin-18 in chronic joint diseases

5. Dysfunctional immune-mediated inflammation in rheumatoid arthritis dictates that development of antirheumatic disease drugs target multiple intracellular signaling pathways;Malemud C. J.;Antiinflamm. Antiallergy Agents Med. Chem.,2011

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4. Reciprocal Alterations in Osteoprogenitor and Immune Cell Populations in Rheumatoid Synovia;International Journal of Molecular Sciences;2022-10-16

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