Regulation of JNK/ERK activation, cell apoptosis, and tissue regeneration by monoamine oxidases after renal ischemia‐reperfusion
Author:
Affiliation:
1. INSERM U388, IFR31, Institut Louis Bugnard, CHU Rangueil Toulouse 31403 Cedex 4 France
2. Department of PathologyCHU Rangueil Toulouse 31403 Cedex 4 France
Funder
Institut National de la Santé et de la Recherche Médicale
Conseil Régional Midi-Pyrénées
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.01-1008fje
Reference59 articles.
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2. Rapid DNA fragmentation from hypoxia along the thick ascending limb of rat kidneys
3. An evaluation of renal tubular DNA laddering in response to oxygen deprivation and oxidant injury;Iwata M.;J. Am. Soc. Nephrol.,1994
4. Regeneration after acute tubular necrosis
5. The stress‐activated protein kinases are major c‐Jun amino‐terminal kinases activated by ischemia and reperfusion;Pombo C.M.;J. Biol. Chem.,1994
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