Hypoxia‐induced deoxycytidine kinase expression contributes to apoptosis in chronic lung disease-Reference-Cited by-同舟云学术

Hypoxia‐induced deoxycytidine kinase expression contributes to apoptosis in chronic lung disease

Published:2013-02-07 Issue:5 Volume:27 Page:2013-2026
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Weng Tingting¹,Karmouty‐Quintana Harry¹,Garcia‐Morales Luis J.²,Molina Jose G.¹,Pedroza Mesias¹,Bunge Raquel R.²,Bruckner Brian A.²³,Loebe Matthias²³,Seethamraju Harish³,Blackburn Michael R.¹

Affiliation:

1. Department of Biochemistry and Molecular BiologyUniversity of Texas–Houston Medical SchoolHoustonTexasUSA

2. Methodist DeBakey Heart and Vascular CenterMethodist HospitalHoustonTexasUSA

3. Methodist J. C. Walter Jr. Transplant CenterMethodist HospitalHoustonTexasUSA

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.12-222067

Reference46 articles.

1. Physical and Functional Interactions between USF and Sp1 Proteins Regulate Human Deoxycytidine Kinase Promoter Activity

2. Adenosine signaling and the regulation of chronic lung disease

3. The hypoxia–inflammation link and potential drug targets

4. Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema;Demedts I. K.;Respir. Res.,2006

5. The Use of Enzyme Therapy to Regulate the Metabolic and Phenotypic Consequences of Adenosine Deaminase Deficiency in Mice

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