The hydrolase DDAH2 enhances pancreatic insulin secretion by transcriptional regulation of secretagogin through a Sirt1‐dependent mechanism in mice-Reference-Cited by-同舟云学术

The hydrolase DDAH2 enhances pancreatic insulin secretion by transcriptional regulation of secretagogin through a Sirt1‐dependent mechanism in mice

Published:2013-02-21 Issue:6 Volume:27 Page:2301-2315
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Hasegawa Kazuhiro¹,Wakino Shu¹,Kimoto Masumi²,Minakuchi Hitoshi¹,Fujimura Keiko¹,Hosoya Koji¹,Komatsu Motoaki¹,Kaneko Yuka¹,Kanda Takeshi¹,Tokuyama Hirobumi¹,Hayashi Koichi¹,Itoh Hiroshi¹

Affiliation:

1. Department of Internal MedicineSchool of MedicineKeio UniversityTokyoJapan

2. Department of Nutritional ScienceOkayama Prefectural UniversityOkayamaJapan

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.12-226092

Reference46 articles.

1. Global Prevalence of Diabetes

2. Mice with gene disruption of both endothelial and neuronal nitric oxide synthase exhibit insulin resistance.

3. Role of nitric oxide synthase isoforms in glucose-stimulated insulin release

4. Expression of islet inducible nitric oxide synthase and inhibition of glucose-stimulated insulin release after long-term lipid infusion in the rat is counteracted by PACAP27

5. A Neuronal Isoform of Nitric Oxide Synthase Expressed in Pancreatic β-Cells Controls Insulin Secretion

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