Nonrandom AP site distribution in highly proliferative cells-Reference-Cited by-同舟云学术

Nonrandom AP site distribution in highly proliferative cells

Published:2006-10-26 Issue:14 Volume:20 Page:2612-2614
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Chastain Paul D.¹,Nakamura Jun²³,Swenberg James²³,Kaufman David¹³,Chastain Paul D.¹,Nakamura Jun²³,Swenberg James²³,Kaufman David¹³

Affiliation:

1. Department of Pathology and LaboratorySchool of MedicineChapel HillNorth CarolinaUSA

2. Department of Environmental Sciences and EngineeringChapel HillNorth CarolinaUSA

3. Curriculum in ToxicologyUniversity of North CarolinaChapel HillNorth CarolinaUSA

Funder

National Institutes of Health

National Institute of Environmental Health Sciences

Southwest Foundation for Biomedical Research

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.06-6145fje

Reference20 articles.

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2. Oxidative DNA damage and disease: induction, repair and significance

3. 5′-Nicked Apurinic/Apyrimidinic Sites Are Resistant to β-Elimination by β-Polymerase and Are Persistent in Human Cultured Cells after Oxidative Stress

4. Endogenous apurinic/apyrimidinic sites in genomic DNA of mammalian tissues;Nakamura J;Cancer Res,1999

5. Highly sensitive apurinic/ apyrimidinic site assay can detect spontaneous and chemically induced depurination under physiological conditions;Nakamura J.;Cancer Res,1998

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