Hypothermia injury/cold‐induced apoptosis—evidence of an increase in chelatable iron causing oxidative injury in spite of low O 2 − /H 2 O 2 formation-Reference-Cited by-同舟云学术

Hypothermia injury/cold‐induced apoptosis—evidence of an increase in chelatable iron causing oxidative injury in spite of low O 2 − /H 2 O 2 formation

Published:2000-10 Issue:13 Volume:14 Page:1953-1964
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Rauen Ursula¹,Petrat Frank¹,Li Tongju¹,De Groot Herbert¹

Affiliation:

1. Institut für Physiologische ChemieUniversitätsklinikumD-45122 Essen Germany

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.00-0071com

Reference51 articles.

1. Involvement of Reactive Oxygen Species in the Preservation Injury to Cultured Liver Endothelial Cells

2. Oxygen-free radical-mediated injury to cultured rat hepatocytes during cold incubation in preservation solutions

3. Injury to Cultured Liver Endothelial Cells After Cold Preservation: Mediation by Reactive Oxygen Species that are Released Independently of the Known Trigger Hypoxia/Reoxygenation

4. Cold-Induced Release of Reactive Oxygen Species as a Decisive Mediator of Hypothermia Injury to Cultured Liver Cells

5. COLD PRESERVATION OF ISOLATED RABBIT PROXIMAL TUBULES INDUCES RADICAL-MEDIATED CELL INJURY1

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