Increased superoxide in vivo accelerates age‐associated muscle atrophy through mitochondrial dysfunction and neuromuscular junction degeneration-Reference-Cited by-同舟云学术

Increased superoxide in vivo accelerates age‐associated muscle atrophy through mitochondrial dysfunction and neuromuscular junction degeneration

Published:2009-12-29 Issue:5 Volume:24 Page:1376-1390
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Jang Youngmok C.¹,Lustgarten Michael S.²,Liu Yuhong¹,Muller Florian L.¹,Bhattacharya Arunabh¹,Liang Hanyu¹,Salmon Adam B.¹,Brooks Susan V.³,Larkin Lisa³,Hayworth Christopher R.⁴,Richardson Arlan¹⁵⁶,Van Remmen Holly¹⁵⁶

Affiliation:

1. Department of Cellular and Structural BiologyUSA

2. Department of PhysiologyUSA

3. Department of Molecular and Integrative PhysiologyUniversity of MichiganAnn ArborMichiganUSA

4. Section of Neurobiology and Institute for NeuroscienceUniversity of TexasAustinTexasUSA

5. Barshop Institute for Longevity and Aging StudiesUniversity of Texas Health Science Center at San AntonioSan AntonioTexasUSA

6. South Texas Veterans Health Care SystemSan AntonioTexasUSA

Funder

American Federation for Aging Research

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-146308

Reference58 articles.

1. Alterations in mitochondrial function, hydrogen peroxide release and oxidative damage in mouse hind-limb skeletal muscle during aging

2. Oxidative stress, chronic disease, and muscle wasting

4. Aging Impairs Skeletal Muscle Mitochondrial Bioenergetic Function

5. What is the cause of the ageing atrophy?

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