CCP1/Nna1 functions in protein turnover in mouse brain: Implications for cell death in Purkinje cell degeneration mice-Reference-Cited by-同舟云学术

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CCP1/Nna1 functions in protein turnover in mouse brain: Implications for cell death in Purkinje cell degeneration mice

Published:2010-01-08 Issue:6 Volume:24 Page:1813-1823
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Berezniuk Iryna¹,Sironi Juan²,Callaway Myrasol B.²,Castro Leandro M.³,Hirata Izaura Y.⁴,Ferro Emer S.³,Fricker Lloyd D.¹

Affiliation:

1. Department of Neuroscience Albert Einstein College of Medicine Bronx New York USA

2. Department of Molecular Pharmacology Albert Einstein College of Medicine Bronx New York USA

3. Department of Cell Biology and Development University of São Paulo São Paulo Brazil

4. Department of Biophysics Federal University of São Paulo São Paulo Brazil

Funder

National Institutes of Health

Financiadora de Estudos e Projetos

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-147942

Reference49 articles.

1. Protein degradation and protection against misfolded or damaged proteins;Goldberg A. L.;Nature,2003

2. Interferongamma can stimulate post‐proteasomal trimming of the N terminus of an antigenic peptide by inducing leucine aminopeptidase;Beninga J.;J. Biol. Chem.,1998

3. The importance of the proteasome and subsequent proteolytic steps in the generation of antigenic peptides;Goldberg A. L.;Mol. Immunol.,2002

4. Metallocarboxypeptidases: emerging drug targets in biomedicine;Arolas J. L.;Curr. Pharm. Des.,2007

5. Purkinje cell degeneration, a new neurological mutation in the mouse;Mullen R. J.;Proc. Natl. Acad. Sci. U. S. A.,1976

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