A central role for inflammation in the pathogenesis of diabetic retinopathy

Author:

Joussen Antonia M.1234,Poulaki Vassiliki34,Le Minh Ly1,Koizumi Kan1,Esser Christina1,Janicki Hanna1,Schraermeyer Ulrich1,Kociok Norbert2,Fauser Sascha2,Kirchhof Bernd12,Kern Timothy S.5,Adamis Anthony P.36

Affiliation:

1. Department of Vitreoretinal Surgery and Angiogenesis LaboratoryCenter of OphthalmologyUniversity of CologneKölnGermany

2. Center for Molecular Medicine (ZMMK) University of CologneKölnGermany

3. Retina Research LaboratoryMassachusetts Eye and Ear Infirmary, Harvard Medical SchoolBostonMassachusetts

4. Laboratory for Surgical ResearchChildren's Hospital, Harvard Medical SchoolBostonMassachusetts

5. Departments of Medicine and Ophthalmology and Center for Diabetes ResearchCase Western Reserve UniversityClevelandOhio

6. Eyetech Research CenterWoburnMassachusetts

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference26 articles.

1. Enhanced expression of intracellular adhesion molecule‐1 and P‐selectin in the diabetic human retina and choroid;McLeod D. S.;Am. J. Pathol.,1995

2. Activated monocytes and granulocytes, capillary nonperfusion, and neovascularization in diabetic retinopathy;Schröder S.;Am. J. Pathol.,1991

3. In vivo demonstration of increased leukocyte entrapment in retinal microcirculation of diabetic rats;Miyamoto K.;Invest. Ophthalmol. Vis. Sci.,1998

4. Prevention of leukostasis and vascular leakage in streptozotocin-induced diabetic retinopathy via intercellular adhesion molecule-1 inhibition

5. Leukocyte-Mediated Endothelial Cell Injury and Death in the Diabetic Retina

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