IRF3 regulates cardiac fibrosis but not hypertrophy in mice during angiotensin II‐induced hypertension-Reference-Cited by-同舟云学术

IRF3 regulates cardiac fibrosis but not hypertrophy in mice during angiotensin II‐induced hypertension

Published:2011-01-25 Issue:5 Volume:25 Page:1531-1543
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Tsushima Kensuke¹²,Osawa Tomoko¹,Yanai Hideyuki¹³,Nakajima Akira¹,Takaoka Akinori¹,Manabe Ichiro²,Ohba Yusuke⁴,Imai Yasushi²,Taniguchi Tadatsugu¹³,Nagai Ryozo²

Affiliation:

1. Department of ImmunologyGraduate School of MedicineUniversity of TokyoTokyoJapan

2. Department of Cardiovascular MedicineGraduate School of MedicineUniversity of TokyoTokyoJapan

3. Core Research for Evolution Science and TechnologyJapan Science and Technology AgencyTokyoJapan

4. Laboratory of Pathophysiology and Signal TransductionHokkaido University Graduate School of MedicineSapporoJapan

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.10-174615

Reference53 articles.

1. Diastolic Dysfunction in Hypertensive Hearts: Roles of Perivascular Inflammation and Reactive Myocardial Fibrosis

2. Patterns of myocardial fibrosis;Weber K T.;J. Mol. Cell. Cardiol.,1989

3. Chymase Inhibition Prevents Cardiac Fibrosis and Improves Diastolic Dysfunction in the Progression of Heart Failure

4. Mechanical Stretch and Angiotensin II Differentially Upregulate the Renin-Angiotensin System in Cardiac Myocytes In Vitro

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